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Leprosy and the adaptation of human toll-like receptor 1. PLoS Pathog (2010)
| Citations: | 4 - 1 self |
BibTeX
@MISC{Wong10leprosyand,
author = {Sunny H. Wong and Sailesh Gochhait and Dheeraj Malhotra and Fredrik H. Pettersson and Yik Y. Teo and Chiea C and Anna Rautanen and Stephen J. Chapman and Tara C. Mills and Amit Srivastava and Aleksey Rudko and Maxim B. Freidin and Valery P. Puzyrev and Shafat Ali and Shweta Aggarwal and Rupali Chopra and Belum S. N and Vijay K. Garg and Suchismita Roy and Sarah Meisner and Sunil K. Hazra and Bibhuti Saha and Sian Floyd and Brendan J. Keating and Cecilia Kim and Benjamin P. Fairfax and Julian C. Knight and Philip C. Hill and Richard A and Hakon Hakonarson and Paul E. M. Fine and Rameshwar N. K and Bamezai Adrian V. S. Hill},
title = {Leprosy and the adaptation of human toll-like receptor 1. PLoS Pathog},
year = {2010}
}
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Abstract
Leprosy is an infectious disease caused by the obligate intracellular pathogen Mycobacterium leprae and remains endemic in many parts of the world. Despite several major studies on susceptibility to leprosy, few genomic loci have been replicated independently. We have conducted an association analysis of more than 1,500 individuals from different case-control and family studies, and observed consistent associations between genetic variants in both TLR1 and the HLA-DRB1/DQA1 regions
