In animal trials clinical outcome and histopathological damage of the brain tissue due to hypoxia after cardiac arrest was found to be reduced by mild therapeutic hypothermia.(1,2) The clinical milestone trials in 2002 also have found mild therapeutic hypothermia at 32°-34 °C for 12-24 hours

Provision of mild therapeutic hypothermia to survivors of cardiac arrest was demonstrated to improve mortality and neurological outcomes in two recent large randomized trials (HACA 2002, Bernard 2002). Additionally, the 2003 ILCOR and 2005 AHA recommendations state “Unconscious adult patients

therapeutic potential of regulated

and routinely administered resuscitation medications. Case Report: We present a case of idiopathic ventricular fibrillation in a previously healthy 36-year-old man who developed persistent ventricular fibrillation in the setting of mild therapeutic hypothermia and high doses of routine resuscitation medications

Clinical application of mild therapeutic hypothermia after cardiac arrest* Jasmin Arrich, MD; The European Resuscitation Council Hypothermia After Cardiac Arrest Registry Study Group Postresuscitative mild hypother-mia decreases mortality and re-duces neurologic impairmentafter cardiac arrest

in surgery patients. On the other hand, crystalloid hemodilution has been shown to induce a hypercoagulable state. The study aim was to elucidate in which way the induction of mild therapeutic hypothermia by a bolus infusion of cold crystalloids affects the coagulation system of patients with cardiac arrest

Mild hypothermia improved neurological outcome and reduced mortality after cardiac arrest because of ventricular fibrillation The Hypothermia After Cardiac Arrest Study Group.Mild therapeutic hypothermia to improve the neurologic outcome after cardiac arrest. N Engl J Med 2002 Feb 21

1. This protocol outlines the approach for instituting immediate mild hypothermia (core temperature of 33-34 degrees Celsius) in patients presenting in coma following cardiac arrest (outof-hospital and in-hospital. Studies have shown that implementation of mild therapeutic hypothermia seems

a b s t r a c t Aim of the study: Out-of-hospital induction of mild therapeutic hypothermia after cardiac arrest needs easy to use and accurate body temperature monitoring. The aim of the study was to evaluate the best temperature probe position on a specially designed tracheal tube, as compared

Abstract. Patients that have survived Out-of-Hospital Cardiac Arrest usually develop some degree of neurological problems. A common treatment to mitigate neurological damage is mild therapeutic hypothermia (MTH). However, MTH has adverse effects, including arrhythmia. In order to explore